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As such they are qualitativelysimilar to those of directly acting cholinoceptorstimulants. These ? ndingssuggest that mutant p53 may be necessary to allow TGF-beta1 pro-metastaticaction.

Indirectly, volumeoverload and increased t.p.r. Similarly to the WT p53that is activated in response to an oncogenic challenge, factors such as oncogenes,DNA damage, and ROS have been shown to stabilize mutant p53

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Usually, three types are described,i.e., wet, dry, and ? xed ?brotic.